Ni G, Ma Z, Wong JP, Zhang Z, Cousins E, Major MB, and Damania B
Category: Uncategorized
Classification of T-cell activation via autofluorescence lifetime imaging (Links to an external site)
Walsh AJ, Mueller KP, Tweed K, Jones I, Walsh CM, Piscopo NJ, Niemi NM, Pagliarini DJ, Saha K, and Skala MC
Inhibition of DNA-J-HSP70 interaction improves strength in muscular dystrophy (Links to an external site)
Bengoechea R, Findlay AR, Bhadra AK, Shao H, Stein KC, Pittman SK, Daw JAW, Gestwicki JE, True HL, and Weihl CC
Gain-of-function genetic screen of the kinome reveals BRSK2 as an inhibitor of the NRF2 transcription factor (Links to an external site)
Tamir TY, Bowman BM, Agajanian MJ, Goldfarb D, Schrank TP, Stohrer T, Hale AE, Siesser PF, Weir SJ, Murphy RM, LaPak KM, Weissman BE, Moorman NJ, and Major MB
Comprehensive nucleosome interactome screen establishes fundamental principles of nucleosome binding (Links to an external site)
Skrajna A, Goldfarb D, Kedziora KM, Cousins EM, Grant GD, Spangler CJ, Barbour EH, Yan X, Hathaway NA, Brown NG, Cook JG, Major MB, and McGinty RK
The mechanism of high-output cardiac hypertrophy arising from potassium channel gain-of-function in Cantú syndrome (Links to an external site)
McClenaghan C, Huang Y, Matkovich SJ, Kovacs A, Weinheimer CJ, Perez R, Broekelmann TJ, Harter TM, Lee J-M, Remedi MS, and Nichols CG
Phospho-Ser784-VCP is required for DNA damage response and is associated with poor prognosis of chemotherapy-treated breast cancer (Links to an external site)
Zhu C, Rogers A, Asleh K, Won J, Gao D, Leung S, Li S, Vij KR, Zhu J, Held JM, You Z, Nielsen TO, and Shao J
Client processing is altered by novel myopathy-causing mutations in the HSP40 J domain (Links to an external site)
Pullen MY, Weihl CC, and True HL
Plasmodium falciparum translational machinery condones polyadenosine repeats (Links to an external site)
Pavlovic-Djuranovic S, Erath J, Andrews RJ, O Bayguinov PO, Chung JJ, Chalker DL, Fitzpatrick JAJ, Moss WN, Szczesny P, and Djuranovic S
Whole exome sequencing in patients with Williams-Beuren syndrome followed by disease modeling in mice points to four novel pathways that may modify stenosis risk (Links to an external site)
Parrish PCR, Liu D, Knutsen RH, Billington CJ, Mecham RP, Fu Y-P, and Kozel, BA